And when an outbreak occurs, the vaccine still protects. A second CDC study examined an explosion in a Kentucky nursing home in which half of the workers received a full vaccination. The disease, which was started by a non-vaccinated worker, caused 46 covid-19 infections. Of the 71 residents who received the vaccine, 18 (25%) became ill, two were hospitalized, and one died. The staff went well. Of the 56 workers who received the vaccine, four (7%) became infected. Most of these diseases were asymptomatic. Only 6.3% of residents and employees who received the vaccine began to develop symptoms, compared with 32% of those who did not receive the vaccine.
In a wake-up call at the nursing home, “workers and residents are in constant contact with the SARS-CoV-2 HIV virus,” says Meagan Fitzpatrick, a communicator at the University of Maryland School of Medicine. Therefore seeing less disease in this category is encouraging.
Pursuit of colors
New research also suggests that species may be the cause of some of these diseases. The problem is “one of the natural cards,” says Anthony Fauci, senior medical adviser to the president of the United States. in short on April 12. Although there is not much information available in the world, lab research shows that some of these are more susceptible to vaccine antibodies than the first SARS-CoV-2.
In a study in Kentucky, the researchers found that the explosion was caused by a type of R1, which is still unknown in the state. The problem was with a number of important variables found in other species. For example, mutations in E484K, which are also found in the first version of B.1.351 in South Africa, appear to be helping the virus to fight off antibody responses. And changes to the D614G can increase mobility. The authors note that although the vaccine reduced the risk of transmission and infectious disease, the virus was still able to infect more than a quarter of the vaccine population and about 7% of the workforce. This suggests that the vaccine may not work against this, but the authors warn that the study was limited. (Chicago authors did not comment on the virus.)
A study by the New England Journal of Medicine diagnosed the disease at staff at Rockefeller University in New York. Between January 21 and March 17, the researchers tested 417 employees who received the complete Pfizer or Moderna vaccine. Two women were infected. When the researchers tested the viruses, they found that each one was slightly different, and it was not exactly the same as what was already known.
For example, one woman had some changes found in B.1.1.7, which came from the UK, as well as changes known as B.1.526, which came from New York City. “They had some differences between the two,” said Robert Darnell, a physician and scientist at Rockefeller and lead author of the study.
When the disease started, it was thought that the patient had failed to receive the vaccine, Darnell says. But that was not the case with the woman. Darnell was able to have his blood tested as soon as he was tested. He and his friends discovered several antibodies that could affect SARS-CoV-2. Because she was infected with the virus, her response was due to the vaccine, not her recent illness. Antibodies take time to develop.
The reason why her immune system does not protect her from infection is not clear, but one possibility is that the differences were able to respond to her response. “For this patient, then that’s why we’ve seen so well,” says Stephen Kissler, a specialist at TH Chan School of Public Health at Harvard. “It’s no surprise to me that most of the outbreaks we see are from the nations,” he added. The more people who receive a vaccine, the more difficult it is to choose which ones to use.
On the other hand, when more people are vaccinated, we will see fewer infections and the virus has less chance of mutation. And Fitzpatrick points out that while running out of body describes a woman’s illness, it’s only one case. And there is no evidence that he passed it on to others who received the vaccine. This surprise is worth studying in the future, but “I don’t see this as dangerous,” he says. “There are still public health problems.”
And even if the disease starts, it doesn’t mean the vaccine has failed, says Monica Gandhi, an infectious disease specialist at the University of California, San Francisco. Antibodies are just one part of the immune system. T cells also play a key role in invading other immune systems and removing one virus from the body. They do not protect against infection, but they can spread the virus. And some studies suggest that the immune response to T cells may be more difficult to avoid. “You may have a few illnesses, but I hope you will be immune to dangerous diseases,” Gandhi said.
However, it is important to follow preventive measures in order to detect any sudden changes. The prevalence of the disease in people who are vaccinated may mean a decrease in immunity or the emergence of a new type of immunodeficiency virus. The vaccine may need to be adjusted, and we may need to shoot. But over time, “our bodies develop a complete immune system,” says Kissler. “And even if we do, we will be protected from the worst possible consequences. In time, his attitude softened. ”