This Is Your Brain Under Anesthesia
In previous studies it fills the brain in rats and EEG readings from people, Brown pointed out that propofol interferes with contact with the cortex. But in order to advance science, he and Miller wanted to draw different parts at the same time as the animal that enters and exits. They wanted to use plant electrons to listen to neurons that change their tones to find out how, and that – difficult brain connections are terminated by anesthesia. In their new research, they developed 64-channel microelectrodes in four rhesus macaque monkeys. These were connected to four parts of their brain: the three parts of the cortex and the thalamus. The three areas of this type are the frontal, temporal, and parietal lobes, which are connected by perception, auditory processing, and sensory perception, respectively. The thalamus is about the size of a quail’s egg and lives inside the brain, sending more information around the cortex.
Scientists hit Record on the electrodes before using the first propofol, only to see the monkeys faint. “The drug goes everywhere, and it goes there in seconds,” Brown said. Brain waves slowed crawling. (Neurons in a healthy, alert brain come out 10 times a second. Under propofol, the bones fall out once a minute or less.) Brown was not surprised; he had seen these species slowly move into other animals, including humans. But deeper electrons can now answer something more accurately: What is happening between neurons?
In most cases, neurons chitchat in combination. “I feel like an FM radio,” Miller says. “In one way or another, they can communicate.” Millions of neurons communicate this way, often in different ways. But now, the constant frequency of frequency has been changed to one short-lived one – one amazing connection. The higher frequencies were gone, and the neurons were left communicating on a much smaller channel. It’s like the sound of a dining room full of children talking in large groups, one quiet one, and everything in between, just ruined.
According to Brown, fewer spikes of neural events during anesthesia are more closely related than in all other cases. Whether you are awake, reading, sleeping, or meditating, your brain is fragile and difficult to monitor. But there is no clear and clear indication for EEG as anesthesia. And, deeply, he believes that, these similarities reduce awareness. The communication of the dining room from the cerebral brain seems like a distraction, but it is a common language of memories, thoughts, and feelings. The sound of anesthesia is evident, but it is a wilderness of knowledge.
“Propofol comes in like a bear,” Miller says, “and he just knocks his brain out in a way that is impossible when he can’t.”
Miller and Brown thought the thalamus would be crucial in reversing the wake-up call. Another existing theory suggests that, in order to produce information, this small nub is compatible with a variety of codecs. If the thalamus stops functioning, the theory states, the fast-moving waves cannot match their speech to express a coherent idea. “And the connection is Everything knowingly, ”says Miller.
After noticing that anesthesia has reduced connectivity from the thalamus, the researchers wanted to see if stimulating the brain area could produce symptoms. Previous work has shown that deep brain stimulation can restore the normal range of motion of a person with a severe brain injury, as well as their ability to eat. However, the idea is new. “It was a little sniffing, a long shot,” Miller says.